Fig. 2From: Molecular epidemiology and hematologic characterization of δβ-thalassemia and hereditary persistence of fetal hemoglobin in 125,661 families of greater Guangzhou area, the metropolis of southern ChinaStructures of the common deletions and the locus of the (Aγ-196 C-T) mutation identified, LCR, locus control regionBack to article page