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Table 1 Clinical and laboratory features of 47 patients suffering from PCOS and 45 controls included in this genetic study

From: Abnormal glucose tolerance and insulin resistance in polycystic ovary syndrome amongst the Taiwanese population- not correlated with insulin receptor substrate-1 Gly972Arg/Ala513Pro polymorphism

Characteristics PCOS (n = 47) Normal (n = 45) P
Age (years) 26.3 ± 5.4 24.6 ± 3.9 NS
Acanthosis nigrans (%) 31.9% 0% <0.05
Body weight (kg) 72.4 ± 14.5 56.4 ± 9.8 <0.05
BMI (kg/m2) 28.5 ± 6.0 21.7 ± 3.2 <0.05
LH/FSH 2.1 ± 1.0 1.3 ± 0.6 <0.05
Prolactin (ng/ml) 18.6 ± 8.0 12.7 ± 6.3 NS
Testosterone (ng/ml) 1.3 ± 0.5 0.5 ± 0.2 <0.05
SHBG 21.1 ± 8.3 48.1 ± 7.8 <0.05
Fasting glucose (mg/dl) 91.3 ± 18.1 86.3 ± 17.6 NS
IGT (%) 36.2% 6.25% <0.05
AGT (%) 46.8% 6.25% <0.05
Fasting insulin (uU/ml) 24.4 ± 17.2 7.9 ± 6.7 <0.05
Glucose/insulin ratio 5.27 ± 3.34 12.13 ± 4.94 <0.05
HOMAIR 5.21 ± 3.73 1.59 ± 0.65 <0.05
  1. PCOS = polycystic ovary syndrome; BMI = body mass index; SHBG = sex hormone binding globulin; IGT = intolerance of 75 gm oral glucose tolerance test; Glucose/insulin ratio = fasting glucose (mg/dl)/fasting insulin (uU/ml); AGT = abnormal glucose tolerance; HOMAIR(Homeostasis model assessment index for insulin resistance) = fasting insulin (uU/ml) × fasting glucose (mg/dl)/405